Thursday, November 28, 2019

A History of African Slave Traders

A History of African Slave Traders During the era of the trans-Atlantic slave trade, Europeans did not have the power to invade African states or kidnap African slaves at will. For the most part, the 12.5 million slaves transported across the Atlantic Ocean were purchased from African slave traders. It is a piece of the triangle trade about which there are still many critical misperceptions. Motivations for Slavery One question that many Westerners have about African slavers, is why were they willing to sell their own people? Why would they sell Africans to Europeans? The simple answer to this question is that they did not see slaves as their own people. Blackness (as an identity or marker of difference) was a preoccupation of Europeans, not Africans. There was also in this era no sense of being African. (Indeed, to this day, individuals are more likely to identify as being African rather than, say, Kenyan only after leaving Africa.) Some slaves were prisoners of, and many of these may have been seen as enemies or rivals to those who sold them. Others were people who had fallen into debt. They were different by virtue of their status (what we might think of today as their class). Slavers also kidnapped people, but again, there was no reason they would inherently see slaves as their own. Slavery as a Part of Life It might be tempting to think that African slave traders did not know how bad European plantation slavery was, but there was a lot of movement across the Atlantic. Not all traders would have known about the horrors of the Middle Passage or what life awaited slaves, but others at least had an idea. There are always people willing to ruthlessly exploit others in the quest for money and power, but the story of the African slave trade goes much further than a few bad people. Slavery and the sale of slaves, though, were parts of life. The concept of not selling slaves to willing buyers would have seemed strange to many people up until the 1800s. The goal was not to protect slaves, but to ensure that oneself and ones kin were not reduced to slaves. A Self-Replicating Cycle As the slave trade intensified in the 16 and 1700s, it also became harder not to participate in the trade in some regions of West Africa. The enormous demand for African slaves led to the formation of a few states whose economy and politics were centered around slave raiding and trading. States and political factions that participated in the trade gained access to firearms and luxury goods, which could be used to secure political support. States and communities who were not actively participating in the slave trade were increasingly at a disadvantage. The Mossi Kingdom is an example of a state that resisted the slave trade until the 1800s when it began trading in slaves as well. Opposition to the Trans-Atlantic Slave Trade The Mossi Kingdom was not the only African state or community to resist selling slaves to Europeans. For instance, the king of the Kongo, Afonso I, who had converted to Catholicism, tried to stop the slave of slaves to Portuguese traders. He lacked the power, however, to police the whole of his territory, and traders as well as nobles engaged in the Trans-Atlantic slave trade to gain wealth and power. Alfonso tried writing to the Portuguese king and asking him to stop Portuguese traders from engaging in the slave trade, but his plea was ignored. The Benin Empire offers a very different example. Benin sold slaves to Europeans when it was expanding and fighting many wars - which produced prisoners of war. Once the state stabilized, it stopped trading slaves, until it started to decline in the 1700s. During this period of increasing instability, the state resumed participation in the slave trade.

Monday, November 25, 2019

Technical Description Definition essays

Technical Description Definition essays The word distributor can be defined into three different meanings. The first meaning, one that distributes, is the easiest to see since distribution comes from the word distribute. Distribution can also be defined as a wholesaler, or one that markets a commodity. The last definition of distribution is an apparatus in the ignition system that distributes the proper amount of electrical voltage to each spark plug at the correct sequence and time. The word is form the 15th century and it is pronounced di- stri-by Every automobile from todays era has a distributor. The distributor is the major component of the ignition system. The distributor is on either the side or the top of your engine, or up near the firewall (Crouse 304). The distributor looks somewhat like an octopus since it has wires that come out from the top of the cap. The distributor is designed to make and break the ignition primary circuit, a low-tension circuit, and to distribute the resultant high voltage to the proper cylinder at the correct time. The high voltage comes from the coil. The coil is a transformer outside of the distributor that changes low-voltage energy into high-voltage energy. After the high voltage comes out from the coil, it passes to the center terminal of the distributor cap and down the rotor by a wire attached to the coil that goes into the center tower of the distributor cap (Crouse 305-306). The purpose of the cap and rotor is to distribute the spark energy to the proper plug. The distributor cap covers the top part of the distributor. The cap has a tower for each spark plug cable, plus a center tower for the wire from the coil. The number of towers depends on the number of spark plug cables which is the same as how many cylinders the vehicle has. So a four-cylinder vehicl ...

Thursday, November 21, 2019

Comparison of solar deities, specifically Ra, Surya, Apollo Essay

Comparison of solar deities, specifically Ra, Surya, Apollo - Essay Example stic religions and enjoyed much respect and honor and were depicted in the religious scriptures with their great deeds, relations with other deities and mortal people, myths and so on. However, they belong to different cultures: Apollo is a Greek god of Olympic period, Ra is a supreme solar deity of Old Kingdom period, and Surya is a Hindu deity referring to the sun (Gopal, 1990, p.76) and embodying the so-called â€Å"supreme light†. All three deities are male and very powerful, but Ra is granted more power as the creator. Ra is believed to have created himself and to be the supreme deity in ancient Egyptian religion, while Apollo was born from Zeus and Leto and Surya is the son of Dyaus Pita and Aditi (both are sons of the supreme gods of the pantheons, yet some sects consider Surya the Supreme Deity). Both Apollo and Surya have siblings: Apollo’s antipode and the lunar deity is his twin sister Artemis, and Surya is considered one of seven brothers Adityas, the cosmic deities. One more distinction of Ra is the form of his portrayal: Surya and Apollo are entirely anthropomorphic deities depicted both in sculpture and in iconography as humanlike beings; while Ra has many guises and can be depicted as a calf, child, lion or a man with the head of a falcon. The similarity to be highlighted between Apollo and Surya is their main attributes – both have chariots, yet Apollo’s one drawn by white swans and Surya’s chariot is horse-drawn (seven horses embodying sunrays or colors of rainbow). Ra, on the other hand, does not have such an attribute, having a sun disk, a scepter and uraeus, a serpent placed on his crown. Ra and Apollo’s worship cults are characterized by certain centers of worship and places of origin. For Ra, the place of origin and the principal center of worship is Heliopolis, while Delphi is the center of Apollo’s cult, where the temple with his oracles was located. The principal peculiarity of Surya in comparison to the other two deities

Wednesday, November 20, 2019

Research Methodology Paper Example | Topics and Well Written Essays - 2500 words

Methodology - Research Paper Example Secondary research and selection process Secondary research is the process of gathering pertinent information from the work of various scholars. The data collection technique accompanies additional methods to ensure accuracy in the development of the research findings. The technique gives researchers credible basis and guidelines to improve on. Secondary sources of data include electronic resources, books, journals and appropriate publications. The resources must carry relevant information to realize the research objectives. These are identifiable based on the researcher’s judgment that determines their reliability. In this study, the researcher has identified six journals to facilitate data collection. A systematic selection procedure sample (SSPS) that ensures the use of reliable resources was instrumental in the identification of the journals. Outline of six journal articles The journals are chosen as secondary data gathering techniques based on their relevance. They provid e appropriate information after a systematic study that led to the identification of the impact of tourism in fostering peace and growth. The journals enable the realization of valid and reliable results with the capacity to influence decision-making. Paulo (2002) who is a renowned writer explores the benefits of tourism in the society. The author focused on the benefits of tourism with an aim of relaying credible information to diverse stakeholders in the industry. This is with the intention of advancing the practice. He stated that tourism is significant in shaping the economic soundness of individuals and nations. Nations that focus on growth are responsible for adopting noble tourism practices to enhance monetary and social integration. The information also equips policy makers with the information highlighting the benefits of tourism. This enables them to formulate appropriate guidelines to drive operations in the sector. This is critical since the benefits of tourism are achie vable through the development of logical polices that are economically viable. Tourism presents fiscal, cultural, ecological, political and communal benefits. The benefits lead to the improvement of economic performance, rise in the living standards of the locals and cultural integration. These elements are fundamental in developing cohesive communities that are economically vibrant. In particular, tourism contributes to the creation of jobs to the citizens; furthermore, it improves income earnings, conservation of resources and restoration of cultural practices. It fosters understanding between individuals and community members through systematic engagement that leads to cohesiveness. The practice also contributes to cultural understanding, tolerance and protection of the national heritage. As noted by Paulo (2002) tourism enables the community members to enjoy the natural resources and facilitates understanding of the historical events that shape individuals future dealings. There fore, tourism presents immense benefits that nations should adopt. This would facilitate economic growth and social amalgamation. Bull (1999) focuses on the role of tourism in the economic development in the contemporary society. The author stated that tourism influences economic performance in various settings since it contributes to the GDP or the income per-capita. The income generated by the industry contributes significantly in transforming individual’

Monday, November 18, 2019

The Federal Government's Power through Application of the Commerce Research Paper

The Federal Government's Power through Application of the Commerce Clause - Research Paper Example The constitution however, does not provide an outright explanation for the term commerce. What then can be said to constitute commerce? Over the years commerce has been defined to mean the buying and selling of goods across states, this is known as interstate commerce and within states, which is known as intrastate commerce (Barnett, 2001). In Carter vs. Carter Coal Company1, the Supreme Court defined commerce as intercourse with the main reason of trade. Acts that fall within the concept of commerce are those acts that have a substantial effect to the exchange of goods within and across states. As seen under the commerce clause the federal government has the power to regulate commerce. What does it mean to regulate commerce? Regulating commerce according to Chief Justice Marshall regarding the case of Gibbons .v. Ogden2 means having the power to control the manner in which commerce is to be governed. Chief Justice Melville Fuller also gave a similar definition by stating regulation of commerce to mean the authority to prescribe the set of rules outlining the way the governing of commerce should take place. The federal government is formed out of a union between states and the sovereign power and this power is divided is therefore split between the national government and the various states (Bork & Troy, 2002). Discussion The original need for the commerce clause was mainly to discourage rivalry between those states that had a commercial advantage and those that did not have a commercial advantage. This difference in commercial advantage of states was brought due to the proximity to the harbor. Those states that were close to the harbor had a commercial advantage than those that were not. The commerce clause was initially meant to ensure that United States of America was a place where trade could take place freely without states having to bring up barriers against each other (Forte, 2011) therefore giving rise to rivalry. Originally, the term to regulate was in terpreted as making something regular. This clause was intended to make all the states regular by promoting activities that would encourage free trade across and within states and also to oversee any interference in interstate commerce. Federal power was intended to operate activities that fell within the scope of commerce but the courts kept quiet even when the federal government continued to see the general extension of the commerce clause. A good example is the case of Kidd vs. Pearson3 the court recognized the broad powers of the congress under the commerce clause over the sale of good in interstate commerce. The commerce clause was dormant over the years until the courts started making decisions that differentiated commerce and production (Bork & Troy, 2002) as seen in the case of Carter vs. Carter Coal Company4 the Supreme Court defined commerce as the intercourse with the main purpose of trade. The turning point of courts decisions came in with the landmark case of US vs. Lop ez5 the attempt of the federal government to regulate activities that are not commercial was struck down in a ruling. The court stated that the power of the congress under the commerce clause is limited to only commerce and the power to regulate the carrying of guns was not commercial

Friday, November 15, 2019

Sudden Sensorineural Hearing Loss (SSNHL) Intervention

Sudden Sensorineural Hearing Loss (SSNHL) Intervention Henry Davis Hearing loss is not a common healthcare issue addressed by Nurse Practitioners (NP) and Sudden Sensorineural Hearing Loss (SSNHL) occurs even less frequently. Often the onset of hearing loss (related to aging or noise exposure) is insidious, often spanning months to years and is typically bilateral. Hearing loss that occurs with advanced age is routinely compensated for with the use of electronic devices that amplify sound and this type of hearing loss is typically not cured. SSNHL by contrast has a well-defined acute onset within 3 days and is characteristically unilateral hearing loss. Some cases of SSNHL could be cured if a diagnosis is made and treatment initiated within a short time from onset of the hearing loss (Raghunandhan et al., 2012). Nurse practitioners may often be the first healthcare provider the patient encounters. Because of the early patient contact, nurse practitioners are uniquely poised to initiate the first line treatment after consulting with neurology, but prior to referral for follow-up care and further evaluation. Recent research supports that patients who receive early medical treatment for SSNHL recover hearing more often than those, who received delayed care (Raghunandhan et al., 2012). Epidemiology Current research indicates the morbidity of SSNHL is 2-20/ 100,000 persons annually and the mean age of occurrence is 43-53 years old. Mortality as not reported other than a possible link between SSNHL and strokes. Gender does not appear to influence incidents. Risk factors for SSNHL include; advanced age, concurrent cardiovascular disease and the presence of a positive antinuclear antibody (Weber, 2014). One study indicated â€Å"†¦viral infection being the most common etiological factor† for SSNHL (Raghunandhan et al., 2012, p. 229). Etiology The cause of SSNHL is currently considered idiopathic, however the preponderance of current research is directed at the supposition that the etiology of SSNHL is due to edema of the eighth cranial and subsequent compression of the internal auditory artery to the cochlea and circular apparatus. The likely cause of the compression is edema of the auditory nerve within the confined space of the internal auditory meatus from a viral infection (Mom, Chazal, Gabrillargues, Gilain, Avan, 2005). There is minimal clearance for the nerves and vessels. If any edema occurs, compression of the associated structures can result. This would be similar in nature to the pathology associated with Bell’s palsy or compartment syndrome associated with a casted extremity. The surrounding tissue swells and compresses adjacent structures. Cause of SSNHL is currently identified as idiopathic but current treatment targets a viral infection as the causative agent. Early treatment with steroids and antiviral drugs demonstrated reduction in percentage of patients who experience total hearing loss. The nurse practitioner can begin treatment of the patient and ensure timely neurologic specialty follow-up and magnetic resonance imaging (MRI) to rule out acoustic neuroma (Chen, Halpin, Rauch, 2003). Pathogenesis Initially to understand SSNHL we first should examine the anatomy and function of the inner ear. The ear communicates with the brain via eighth cranial nerve. The eighth cranial nerve divides into two main branches, cochlear and vestibular nerves. The first cochlear nerve travels to the cochlea and the vestibular nerve travels to the semicircular ducts. The Cochlea is the portion of the inner ear responsible for the transformation of sound from a physical force, from the tympanic membrane via the malleus, incus and stapes into a nerve impulse conducted to the brain via the cranial nerve (Copstead Banasik, 2013). The vestibular nerve conducts impulses from the semicircular ducts. Semicircular ducts change the forces of air movement into fluid movement that in turn is changed into nerve impulses that allow for proprioception. Damage to this branch of the vestibular nerve can result in vertigo, nystagmus, vomiting and disruptions of proprioception (Copstead Banasik, 2013). The eighth cranial nerve passes through a relatively small opening, the internal auditory meatus, in the skull(Weber, 2014) (Kim Lee, 2009). This small opening also provides the arterial blood flow for the inner ear via the internal auditory artery (Kim Lee, 2009). Even a small amount of edema in the eighth cranial nerve can result in both compression of the nerve and occlusion of the arterial blood supply to the inner ear. The result of these Sequelae would be rapid hearing loss, nystagmus and vertigo (Weber, 2014). During the acute period, the first few hours, it would be possible to decrease the edema by administration of steroids thereby decompressing the nerve and re-establishing blood flow to the inner ear (Narozny et al., 2006). Delays in the reduction of this edema may result in tissue necrosis and clot formation due to stasis of blood in the internal auditory artery. Another less researched cause of SSNHL is vertebrobasilar ischemic stroke or a thrombi occlusion of the arterial supply to the inner ear (Kim Lee, 2009). The resultant Sequelae following the occlusion would mirror the presentation of SSNHL from edema however; the key difference would be the response to oral steroids is ineffective. Any negative effects of oral steroids, if an ischemic stroke were diagnosed would be minimal as the follow-up for MRI should be performed immediately after initial treatment. Clinical Manifestations Onset of SSNHL appears as idiopathic and not related to any trauma and patient denies current source of infection. The patient may report having a clogged ear or pressure in ear. There is an absence of signs and symptoms of infection such as fever, drainage and pain. The patient may find it difficult to impossible to determine the direction of the source of a sound, as both ears are needed to identify the direction of a source of a sound. The brain normally uses the auditory input from both ears identify source location of an auditory stimulus. The occlusion of blood flow to the cochlea on one side has resulted in complete hearing loss to that side. SSNHL may present with an acute onset of tinnitus in the affected ear. The patient may describe the sound as a ringing or roaring sound. Initial onset of tinnitus may cause difficulty in concentration. The pathology responsible for tinnitus remains unclear. The onset of SSNHL is rapid and unilateral, proceeding from normal hearing to complete absence of hearing of the effected ear in less than 3 hours(Weber, 2014). SSNHL is often accompanied by nystagmus, tinnitus and vertigo. This may be due to the compression of the internal auditory artery and the resultant occlusion of blood flow to the cochlea and semicircular apparatus. In addition, when we consider the functions of the central nervous system in the perception of proprioception, multiple stimuli typically confirm the position of the body such as the semicircular apparatus and visual stimuli. With the sudden cessation of blood flow to the inner ear, a mismatch of stimuli rapidly occurs. This mismatch of stimuli among various nerves results in the perception of vertigo and often nystagmus. The Performance of a Rinne test and Weber test enables the nurse practitioner to differentiate between bone conduction damage, as is the case with traumatic injury or nerve damage as is the case in both SSNHL and ischemic stroke (Tintinalli, 2010). Also important is the Dix-Hallpike test, to differentiate between central or peripheral vertigo (Furman Barton, 2014). Central vertigo would be those with a site of origin inside the brain, were as peripheral vertigo originates outside the brain. The Dix-Hallpike test differentiates between central and peripheral vertigo. A Dix-Hallpike test is positive if vertigo and nystagmus is elicited when the head is rotated. Dix-Hallpike test also identifies unilateral vertigo, as well as it differentiates between central and peripheral causes of vertigo and nystagmus. Benign positional nystagmus is also evaluated utilizing the Dix-Hallpike test. In (BPN) the result is a delay of onset of nystagmus of >20seconds, nystagmus slowly resolves if head h eld in the same position, and response decreases with repeated testing (Dix-Hallpike Test, 2014). A central cause of vertigo and nystagmus need to be evaluated if the Dix-Hallpike test yields atypical results of nystagmus that occurs without rotation of the head, begins without delay, and does not decrease with retesting(Dix-Hallpike Test, 2014). Treatment The recommended treatment of SSNHL is early administration of oral glucocorticoids (Raghunandhan et al., 2012)(Narozny et al., 2006). This intervention is well within the scope of practice of the Nurse Practitioner. Nurse Practitioners should be encouraged to take the lead in diagnosing and initiating treatment for SSNHL. The risk associated with early treatment of suspected SSNHL is minimal however if treatment is delayed hearing loss may be permanent. The indication for administration of steroids is to decrease edema of the eighth cranial nerve as this edema leads to loss of nerve conduction and ultimately nerve tissue death and possible arterial occlusion (Chen et al., 2003). The result of tissue death of the eighth cranial nerve is hearing loss, nystagmus, tinnitus and impaired balance (Copstead Banasik, 2013). Current treatment of SSNHL aims at the reduction of damage to the eighth cranial nerve. Other medications can be used to treat the after effects of SSNHL such as vertigo. Select antihistamines, which are also classified as vestibular suppressant, are often used to decrease the perception of vertigo. These drugs reduce â€Å"the activity in the vestibular nuclei and cerebellum.†(Denner, 2013, para. 6). This class of medication is used as needed for vertigo control. Vestibular suppressant antihistamines are first line medications for vertigo. If Vestibular suppressant antihistamines are not successful in controlling vertigo then an escalation to a low dose benzodiazepine most often resolves vertigo not controlled by first line medications. Benzodiazepines cause central nervous system depression and thereby decrease vertigo. The treatment of choice for chronic vertigo is vestibular rehabilitation. Vestibular Rehabilitation is an exercise-based therapy used to retrain the central nervous system (CNS). Often vestibular rehabilitation is sufficient to control symptoms of vertigo without the need for daily medications. The provider often refers the patient experiencing vertigo to physical therapy (PT) or occupational therapy (OT) for a course of progressive exercises with associated head/eye movements to retraining the CNS (Vestibular Rehabilitation Therapy (VRT), 2014). Vestibular rehabilitation can greatly reduce the need for medications to reduce symptoms of vertigo and improves the quality of life. Vestibular rehabilitation is only the first of many topics that should be addressed by the nurse practitioner to assist the patient in lessening symptoms and coping with functional loss. Patient education is targeted toward identification of limitation and practices to compensate for those limitations. Simple functions such as body position during sleep can cause functional problems for patients. An example would be if patient sleeps on the non-affected ear toward the pillow, they would not hear sounds such as alarm clocks, smoke alarms, or other auditory products. Specialized alarms are available which provide both auditory and tactile stimuli (a strong bed vibrating unit) to awaken non-hearing or limited hearing persons. Also, educate patient that stressful situations and loud environments can exacerbate the experience of tinnitus and vertigo. Family members of the patient would be educated on actions to compensate for patient’s hearing loss. Walking on the non-affected side and not speaking close to affected ear are two examples. Also explaining the emotional stressors for the patient and the family members as both learn to cope with the new limitations. The Nurse Practitioner can also provide a referral to an ear, nose and throat specialist to evaluate the patient for cochlear implant for complete unilateral hearing loss. Current implants consist of a base magnet implant into the mastoid bone on the affected side. An external device is worn over the magnet and conducts sound into the magnet and across to the functional ear via bone conduction. The delay between air and bone conduction allows the patient to regain the ability to directionalize sound stimuli. The negative impacts of this option are cost, some insurances do not cover this surgery or only cover a portion of the total cost and this is a surgical intervention and all invasive procedure have associated risk factors. Conclusion Nurse Practitioners should broaden the scope of examination and treatment when assessing patients with sudden hearing loss. In the face of a presumptive diagnosis of SSNHL, the provider may improve outcomes by beginning treatment prior to conclusive diagnosis. Increased early intervention for SSNHL by Nurse Practitioners and beginning steroid treatment within the first few hours after onset of hearing loss may decrease the incidence of permanent hearing loss. The Nurse Practitioner is able to discriminate between sensorineural and conductive hearing loss. A causative factor is then diagnosed to direct care. SSNHL is an infrequently occurring health problem that can be treated with an early presumptive diagnosis. If the initial diagnosis of SSNHL is delayed for MRI to rule out Acoustic Neuroma, it increases the chance that hearing loss will become irreversible. The initial treatment with oral steroid is relatively low risk, yet this option is often omitted despite the apparent benefit. Although the confirmation of SNNHL requires MRI to rule out a differential diagnosis of acoustic neuroma, steroid treatment could begin as a protective measure. If an Acoustic Neuroma is diagnosed the steroid therapy could be discontinued. The impact of steroid use associated with Acoustic Neuroma has not been examined in current research. Appendix Figure 1 (Arora, 2012, figure 5) Figure 2 (Kim Lee, 2009, figure 2) References Arora, R. (2012). Vestibular Rehabilitation: An Overview. Int J Otorhinolaryngol Clin, 4, 54-69. Retrieved from http://www.jaypeejournals.com/eJournals/ShowText.aspx?ID=3564Type=FREETYP=TOPIN=_eJournals/images/JPLOGO.gifIID=280isPDF=NO Chen, C., Halpin, C., Rauch, S. (2003). Oral Steroid Treatment of Sudden Sensorineural Hearing Loss: A Ten Year Retrospective Analysis. Otology Neurotology, 24, 728–733. Retrieved from http://www.researchgate.net/publication/9088236_Oral_steroid_treatment_of_sudden_sensorineural_hearing_loss_a_ten_year_retrospective_analysis/links/00b7d51c062542efbc000000 Copstead, L., Banasik, J. (2013). Pathophysiogology (5th ed.). St. Louis, MO: Elsevier. Denner, K. (2013). Meclizine – Does it help? Retrieved from http://vestibular.org/news/10-07-2013/meclizine-–-does-it-help Dix-Hallpike test – Quick guide. (2014). Retrieved from http://www.ncuh.nhs.uk/our-services/dix-hallpike-test-quick-guide.pdf FM Jr, B. (1984). Sudden hearing loss: eight years experience and suggested prognostic table. The Laryngoscope, 94, 647-61. Retrieved from http://ezproxy.okcu.edu:2192/ehost/detail/detail?vid=1[emailprotected]hid=4201bdata=JnNpdGU9ZWhvc3QtbGl2ZQ==#db=mnhAN=6325838 Furman, J., Barton, J. (2014). Evaluation of the patient with vertigo. Retrieved from http://www.uptodate.com/contents/evaluation-of-the-patient-with-vertigo?source=machineLearningsearch=Dix-HallpikeselectedTitle=2~5sectionRank=5anchor=H29#H29 Hearing Loss: A Ten Year Retrospective Analysis. Otology Neurotology, 24, 728-733. Retrieved from http://www.tonybaino.com/otorhino/steroidsssnhl.pdf Kim, J., Lee, H. (2009). Inner Ear Dysfunction Due to Vertebrobasilar Ischemic Stroke. SEMINARS IN NEUROLOGY, 29, 534-540. http://dx.doi.org/10.1055/s-0029-1241037 Mom, T., Chazal, J., Gabrillargues, J., Gilain, L., Avan, P. (2005). Cochlear blood supply: an update on anatomy and function. French Ear, Nose Laryngology, 88, 81-88. Retrieved from http://xa.yimg.com/kq/groups/17470070/1437766444/name/KimJS2009 [Inner Ear Dysfunction Due VB Ischemic Stroke].pdf Our Experience. Association of Otolaryngologists of India, 65, 229-233. http://dx.doi.org/10.1007/s12070-012-0506-9 Our Experience and a Review of the Literature. Annals of Otology. Rhinology Laryngology, 115, 554-558. Retrieved from http://ezproxy.okcu.edu:2192/ehost/pdfviewer/[emailprotected]5vid=16hid=4104 Sensorineural Hearing Loss: Prospective Clinical Research. The Journal of Otolaryngology, 36, 32-37. Retrieved from http://ezproxy.okcu.edu:2192/ehost/pdfviewer/[emailprotected]3vid=1hid=4104 Tintinalli, J. (2010). Emergency Medicine: A Comprehensive Study Guide (7th ed.). New York, NY: Mc Graw Hill. Tintinalli, J. (2010). Tintinalli’s Emergency Medicine: A Comprehensive Study Guide (7th ed.). New York, NY: McGraw-Hill. Tintinalli, J. (2010). Tintinalli’s Emergency Medicine: A Comprehensive Study Guide (7th ed.). New York, NY: McGraw-Hill. Vestibular Rehabilitation Therapy (VRT). (2014). Retrieved November 15, 2014, from http://vestibular.org/understanding-vestibular-disorder/treatment/treatment-detail-page Weber, P. (2014). Sudden sensorineural hearing loss. Retrieved from http://www.uptodate.com/contents/sudden-sensorineural-hearing-loss?source=machineLearningsearch=SSNHLselectedTitle=1~6sectionRank=2anchor=H4#H12 Weber, P. (2014). Sudden sensorineural hearing loss. Retrieved from http://www.uptodate.com/contents/sudden-sensorineural-hearing-loss?source=previewsearch=SSNHLlanguage=en-USanchor=H2selectedTitle=1~6#H2 Yuan-Yuan, L., Zhe, J., Bu-Sheng, T., Jian-ming, Y., Ye-Hai, L., Maoli, D. (2008, January 8). A clinical study of microcirculatory disturbance in Chinese patients with sudden deafness. Acta Oto-Laryngologica, 128, 1168-1172. http://dx.doi.org/10.1080/00016480801901626

Wednesday, November 13, 2019

Alcohol And The Church :: essays research papers

Alcohol And The Church It seems to be that our main questions are, Should we use alcohol and what about those that abuse it? How should the Church deal with those that do drink or should we as a society deal with it? While there is nothing in the Bible that says drinking is a sin, but drunkeness is. I believe as a society we do have a problem with alcohol abuse. But in the same respect I do not feel that the church should judge those who do drink socially, regularly, or abusively. When the time comes everyone will be judged individually by God alone. I feel it is his decision solely to do what is best for all. The church may teach not to use alcohol, but to discriminate against those in the congregation (or even those that are not) that do is not a solution to the problem. I feel in order to get alcohol abuse under control we as a society need to teach our children the risks of using alcohol, not only in the home but in the schools as well. To reach the heart of the problem is to face the problem head on. As a social drinker myself (I put my self in this classification) I don't feel I have an alcohol problem just because I enjoy a drink now and then. I do not abuse it and would never put myself behind the wheel of a car to take the risk of taking someone else's life. You don't need to be drunk to cause an accident, it's been proven in many cases only a few drinks can impair someone's stability. If more thought of this there would be less tragedies on our highways. When it comes to food and there are people starving in the world, when we could help by not converting food grains into alcohol, this should be made more aware to our society. I'm not sure most people are aware of this. It is supposedly our main concern to feed the hungry and shelter the poor. If giving up something that only contributes to loneliness and destruction than ever, because there are more that abuse than those who don't, it seems to me to be a logical solution. I think the best we can hope for in our future and our children's is that we have to